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During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
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Abstract titles should be brief and reflect the content of the abstract.
Central venous occlusion is a major concern in patients requiring long-term hemodialysis access, as it can compromise arteriovenous fistula (AVF) function and lead to venous hypertension. While SAPHO syndrome (synovitis, acne, pustulosis, hyperostosis, and osteitis) is a rare autoinflammatory disorder, vascular complications have scarcely been reported. To our knowledge, bilateral subclavian vein occlusion associated with SAPHO syndrome has not been previously described. We report a unique case in which routine shunt percutaneous transluminal angioplasty (PTA) unexpectedly revealed this rare vascular manifestation, providing new insights into access management in dialysis patients.
An elderly male in his 80s with end-stage renal disease, presumed secondary to nephrosclerosis, initiated hemodialysis via a left femoral vein catheter in December, year X. A left forearm AVF was subsequently created. In February, year X+1, the patient was transferred to our institution for maintenance outpatient dialysis. In August, year X+1, routine ultrasonography demonstrated elevated intravascular pressure in the left basilic vein, raising suspicion of central venous stenosis. Angiography was performed via a sheath inserted from the left basilic vein.
Angiography failed to visualize the left subclavian, brachiocephalic, and superior vena cava, suggesting venous malformation or occlusion. Retrograde flow from the left subclavian vein into the basilic vein bifurcation was observed, and balloon angioplasty at this site resulted in immediate improvement of venous pressure. Contrast-enhanced CT revealed bilateral subclavian vein occlusion, partial absence of the left brachiocephalic vein, and extensive collateral thoracic wall pathways, which were considered sufficient to maintain dialysis access through the left AVF. Review of the medical history revealed pleurodesis performed in October, year X, for unexplained pleural effusion. Taken together, these findings suggested that bilateral subclavian vein occlusion was caused by a combination of thoracic outlet syndrome and chronic pleural inflammation related to SAPHO syndrome.
This case represents, to our knowledge, the first report of bilateral subclavian vein occlusion associated with SAPHO syndrome. It highlights a previously unrecognized vascular complication of this rare disorder and raises the hypothesis that SAPHO-related inflammation may contribute to central venous obstruction. Importantly, the case underscores the need for thorough preoperative central venous imaging, such as CT, before AVF creation in order to prevent postoperative venous hypertension and optimize access strategy. For dialysis access specialists, this case emphasizes that even rare systemic disorders may critically influence vascular anatomy and long-term access outcomes.
