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During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
Please follow the instructions below to input your abstract title.
Abstract titles should be brief and reflect the content of the abstract.
Pregnancy-associated thrombotic microangiopathy (TMA) occurs in 1 in 25,000 births, accounting for 8-18% of all TMA cases. It is necessary to differentiate from thrombotic thrombocytopenic purpura, HELLP syndrome, anti-phospholipid antibody syndrome, atypical hemolytic uremic syndrome (aHUS). However, because these pathogenesis are similar, differentiation is often difficult. Pregnancy-associated TMA can be fatal if it becomes severe, thus early diagnosis and treatment intervention are essential.
Case 1: A 36-year-old female, G1P0, was admitted at 37 weeks gestation due to persistent nausea. Following a failed induction of labor, an emergency Cesarean section was performed. On postoperative day 3, she developed dyspnea along with substantial liver dysfunction, thrombocytopenia, and renal dysfunction, leading to a diagnosis of postpartum fulminant HELLP syndrome. She was successfully treated with plasma exchange (PE) and steroids, achieving improvement in hepatic and renal function, and making a favorable recovery.
Case 2: A 28-year-old female, G1P0, underwent an emergency Cesarean section at 32 weeks of gestation due to placental abruption. Postoperatively, she developed renal dysfunction, thrombocytopenia, and anemia. She presented with oliguria and fluid overload and required the initiation of continuous hemodiafiltration. Given the rapid progression of severe thrombocytopenia and kidney injuries, atypical hemolytic uremic syndrome (aHUS) was clinically diagnosed. Treatment with PE and the complement inhibitor eculizumab was initiated immediately. Subsequently, the platelet counts and kidney injuries were improved, allowing for the discontinuation of hemodialysis.
We encountered two cases of pregnancy-associated TMA. Careful differential diagnosis is essential in pregnancy-associated TMA, especially HELLP syndrome, thrombotic thrombocytopenic purpura, and aHUS. As the severe forms can be life-threatening, early diagnosis and prompt therapeutic intervention are crucial.
