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During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
Please follow the instructions below to input your abstract title.
Abstract titles should be brief and reflect the content of the abstract.
Staphylococcus aureus remains a major cause of peritoneal dialysis (PD) – related peritonitis and is associated with variable clinical outcomes. This study compared the clinical features and outcomes of PD peritonitis caused by methicillin-resistant S. aureus (MRSA) and methicillin-susceptible S. aureus (MSSA).
This was a single-centre, retrospective cohort study that included all episodes of MRSA and MSSA PD-related peritonitis that occurred between January 2013 and July 2024. The primary outcome was to compare clinical outcomes among MRSA and MSSA peritonitis episodes.
A total of 50 peritoneal dialysis patients (10 with MRSA and 40 with MSSA) experienced 64 peritonitis episodes (18 MRSA and 46 MSSA). The mean age was 61 ± 13 years, with no significant difference between the MRSA (59 ± 15 years) and MSSA (62 ± 12 years) groups (p=0.50). The majority were male (66%), Chinese (84%), and on automated PD (88%). Baseline comorbidities, including diabetes mellitus (60% vs 65%, p=0.15) and hypertension (30% vs 20%, p=0.33), did not differ significantly between MRSA and MSSA groups. Clinical presentation showed a trend toward greater severity in MRSA peritonitis, with higher frequencies of fever (29% vs 16%, p=0.25), abdominal pain (83% vs 67%, p=0.10), and hypotension (12% vs 2%, p=0.10), while cloudy effluent was common in both groups (65% vs 63%, p=0.89). Median effluent cell counts at presentation were not significantly different (p=0.23). Clinical outcomes were similar between the groups, with no significant differences in hospitalization (72% vs 72%, p=0.97), cure (72% vs 70%, p=0.83), catheter removal (28% vs 26%, p=0.89), or death (6% vs 9%, p=0.67). On univariable analysis, MRSA infection was not associated with increased odds of hospitalization (Odds ratio [OR]: 0.98; 95 % confidence interval [CI]: 0.26–3.39; p = 0.97), cure (OR: 0.88; 95 % CI 0.26–2.94; p = 0.83), catheter removal (OR: 0.92; 95 % CI 0.27–3.12; p = 0.89), or death (OR: 1.62; 95 % CI 0.17–15.56; p = 0.68).
In this single-centre cohort, while the clinical features of MRSA peritonitis may tend to be more severe upon presentation, the definitive clinical outcomes were comparable to those of MSSA peritonitis. Despite its resistant profile, MRSA infection was not associated with worse rates of hospitalization, cure, catheter removal, or mortality. Larger multicentre studies are warranted to further delineate outcome differences and optimize antibiotic strategies for S. aureus peritonitis.
