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During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
Please follow the instructions below to input your abstract title.
Abstract titles should be brief and reflect the content of the abstract.
The heart and kidneys share a complex bidirectional relationship, whereby dysfunction of one organ can precipitate or exacerbate failure of the other. This interdependence is particularly evident in acute decompensated heart failure (ADHF), where impaired cardiac output, venous congestion, and neurohormonal activation can lead to acute kidney injury (AKI), a condition known as cardiorenal syndrome (CRS). The occurrence of CRS in patients with ADHF is common and clinically significant, as it is associated with greater hemodynamic instability, prolonged hospital stay, higher readmission rates, and increased morbidity and mortality. Furthermore, alterations in renal function during ADHF often complicate the therapeutic approach, limiting the effectiveness of diuretic therapy and making volume management more challenging. Understanding the interplay between cardiac and renal dysfunction is therefore essential for optimizing treatment strategies and improving outcomes in this high-risk population.
This is a single-center, retrospective cohort study, involving all patients admitted to our center for ADHF from 1stDecember 2024 to 31st May 2025. We aimed to determine the prevalence of CRS in ADHF and its associative factors.
A total of 66 patients were recruited, with a mean (± standard deviation) age of 64.6 ± 14.7 years. 53% of them were male and 43.9% were Malay ethnicity. Majority of them were overweight (40.9%) and obese (42.4%) with a median (IQR) body mass index of 27.7 (8.6) kg/m2. The prevalence of CRS was 42.4% in our study population. Using univariate analysis, underlying diabetes mellitus; more advance stages of chronic kidney disease (CKD); the use of beta blocker; recent ADHF, AKI, and hospitalization within six months; high heart failure likert scale; hypoalbuminemia; and anemia are associated with CRS in ADHF upon presentation with the respective p-value of 0.002, 0.004, 0.045, 0.018, 0.010, 0.019, 0.015, <0.001, and 0.048. Multiple regression analysis revealed that a low initial heart failure likert scale of 1 reduced the risk of CRS development by 64.8% (OR 0.352, p=0.027) and hypoalbuminemia increased the incidence of CRS in ADHF by 19.1% (OR 1.191, p=0.023) (Table 1).
CRS is a frequent complication in patients with ADHF. A lower initial heart failure likert scale was protective against the development of CRS, while hypoalbuminemia independently increased its incidence. These findings emphasize the importance of early clinical assessment and monitoring of serum albumin in predicting and preventing CRS among patients with ADHF.
