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During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
Please follow the instructions below to input your abstract title.
Abstract titles should be brief and reflect the content of the abstract.
Preeclampsia, once regarded as a reversible disorder, is now recognized to exert long-term effects on maternal health particularly conditions related to systemic endothelial injury. Increasing evidence suggests that preeclampsia is an underappreciated risk factor for chronic kidney disease. Postpartum monitoring of renal function is frequently neglected among this population. This study aimed to evaluate the association between the presence of maternal organ and placental impairment observed at delivery and the subsequent development of postpartum renal dysfunction in preeclamptic pregnancies.
This hospital-based case-control study included 64 pregnant women diagnosed with preeclampsia and proteinuria. Data were collected between January and December 2024 and included participant characteristics, with particular attention to presence of maternal organ and placental impairment (presence of intrauterine growth restriction [IUGR]) according to ACOG diagnosis criteria for preeclampsia. The assessment of maternal organ impairment focused on the following system: hematologic (platelet level), kidney (creatinine serum level), liver (liver transaminases), pulmonary (presence of pulmonary edema), and cardiac system (presence of heart failure). Renal dysfunction is defined as persistent proteinuria three months postpartum confirmed by urine Albumin-to-Creatinine ratio (uACR) examination. Statistical analyses were performed using the independent t-test, Mann–Whitney test, and Fisher’s exact test, with significance set at p < 0.05.
The presence of maternal renal dysfunction at three months postpartum was observed in 26 participants (40.62%). A total of 11 participants (17.19%) had IUGR, with a mean maternal age of 30.27 ± 3.85 years. In this subgroup, the mean serum creatinine level was 0.91 ± 0.32 mg/dL (z = –1.360; p = 0.174) and the mean ACR was 9.14 ± 11.48 mg/g creatinine (z = –2.518; p = 0.012). Further analysis revealed a significant association between the history of IUGR during pregnancy and postpartum renal impairment (OR = 0.112; p = 0.021).
While IUGR is not known to have direct effect on maternal organs, its presence in preeclamptic patients may exacerbate endothelial stress due to heightened metabolic and circulatory demands during delivery. Validation of these findings will require future prospective and longitudinal studies incorporating longer follow-up durations.