Back
For best output, select "Paper Size" as "A4" and "Margin" as "0" or "None".
To save or print to PDF, please select Print Destination > Save as PDF, enable Background Graphics under "More Settings", then click "Save".
During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
Please follow the instructions below to input your abstract title.
Abstract titles should be brief and reflect the content of the abstract.
Hepatorenal Syndrome–Acute Kidney Injury (HRS-AKI) is a form of renal dysfunction occurring in advanced stages of liver cirrhosis, characterized by renal vasoconstriction without structural kidney abnormalities. Differentiating HRS-AKI from acute kidney injury (AKI) caused by sepsis or hypovolemia is often challenging in clinical practice. The atypical or reversible variant arises when multiple precipitating factors coexist; therefore, application of the 2023 International Club of Ascites–Acute Disease Quality Initiative (ICA–ADQI) criteria is essential to establish an accurate diagnosis.
Observational
A 43-year-old male with chronic hepatitis C presented with jaundice, ascites, and easy fatigability, with no history of hypertension, diabetes, or prior kidney disease. Physical examination revealed stable hemodynamics, jaundice, ascites, and mild peripheral edema. Laboratory findings showed a rapid rise in serum creatinine from 0.97 to 5.9 mg/dL and urea from 20 to 122 mg/dL, accompanied by microscopic hematuria, proteinuria, and a urinary albumin-to-creatinine ratio >300 mg/g. Sepsis markers were elevated (leukocytosis, high C-reactive protein, and procalcitonin). Liver function tests indicated hyperbilirubinemia (Total Bilirubin 47.93mg/dl, Direct Bilirubin 31.19) and increased transaminases (GOT/GPT : 277/152). Chest radiograph showed pneumonia, and abdominal ultrasound revealed liver cirrhosis with normal-sized kidneys. The patient received antibiotics, albumin, terlipressin, and fluid restriction, and underwent hemodialysis to manage severe azotemia. Following infection control and volume correction, renal function improved without further dialysis.
We report a 43-year-old male case of Atypical Hepatorenal Syndrome triggered by sepsis associated with chronic hepatitis C, who experienced reversible renal dysfunction.
