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During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
Please follow the instructions below to input your abstract title.
Abstract titles should be brief and reflect the content of the abstract.
Abdominal aortic calcification (AAC) is a critical complication in patients undergoing maintenance hemodialysis (HD), significantly contributing to increased cardiovascular morbidity and mortality. Early identification of clinical predictors for AAC progression is essential for improving cardiovascular outcomes in this high-risk population. This study aimed to identify the clinical and biochemical predictors for the progression of AAC in HD patients.
A longitudinal study of 102 HD patients was followed over six months. Kauppila’s AAC scores were assessed using lateral lumbar spine X-rays at baseline and at sixth month.The progression of AAC was determined by comparing the results of two separate X-rays. Baseline clinical and demographic information was collected, including age, sex, body weight, height, smoking status, urine output, and comorbidities such as hypertension, diabetes mellitus, cardiovascular disease, connective tissue disease, and malignancy. Data on the underlying cause of ESKD, dialysis vintage and medication use (including antihypertensive, antidiabetic, lipid-lowering agents, phosphate binders, and vitamin D supplements) were also obtained. Biochemical parameters such as serum calcium, phosphate, parathyroid hormone, Vitamin D, lipid profile and inflammatory markers, including C-reactive protein were measured. GNRI score was used to assess nutritional status. Univariate and multivariate logistic regression analyses were used to identify significant predictors of AAC progression.
The proportion of patients with presence of AAC at baseline were 53 (52%), mean score was 2.07 ± 3.71 (range, 0–20). AAC progression 6 months later were 77 (75.5%) and mean AAC score was 3.67 ± 4.84 (range, 0–22). There was a significant AAC progression rate 70 (68.6%) between six months period. In Univariate analysis, patients with AAC progression were more likely to be older, history of diabetes mellitus or CVD, HD vintage more than 12 months, have high levels of phosphate, iPTH, calcium phosphate product, CRP and have low levels of HDL than those without progression. Multivariate analysis confirmed that dialysis vintage (OR: 8.89, 95% CI: 1.36–57.99; P = 0.02) was the statistically significant independent predictor of AAC progression in this study.
Regular monitoring of traditional plus CKD MBD risk factors, inflammatory markers and nutritional status along with periodic imaging of AAC, is crucial for the early identification and management of vascular calcification in HD patients. These findings highlight the importance of targeted interventions to slow AAC progression and improve cardiovascular outcomes in this vulnerable population.
