Back
For best output, select "Paper Size" as "A4" and "Margin" as "0" or "None".
To save or print to PDF, please select Print Destination > Save as PDF, enable Background Graphics under "More Settings", then click "Save".
During the congress, E-Posters will be accessible to all participants on the congress website 24/7, as well as in the E-poster stations in the congress center.
Preparing your E-Poster
Please review the E-Poster format requirements carefully when preparing your E-Poster. Should your E-Poster not meet the mentioned requirements, it may not be displayed as described above.
E-Poster Submission Deadline
Please prepare and upload your E-Poster no later than March 14, 2026 11.59PM CET. After this date, you will no longer be able to prepare and upload your E-poster and it will not be displayed and accessible on the congress website.
Please follow the instructions below to input your abstract title.
Abstract titles should be brief and reflect the content of the abstract.
COVID-19 infection can lead to acute kidney injury and accelerate renal deterioration in patients with pre-existing chronic kidney disease (CKD). Identifying clinical and laboratory factors associated with renal replacement therapy (RRT) initiation and poor outcomes is crucial for early risk assessment.
This retrospective study included 77 patients (median age 67.1 ± 13.7 years) with baseline renal dysfunction who recovered from COVID-19. Demographic, clinical, and laboratory data were analyzed to identify predictors of RRT initiation and mortality. Correlations between biochemical markers and clinical variables were explored using Pearson correlation and regression analysis.
Of the 77 patients, 56 (72.7%) had known CKD, and 32 (41.6%) required RRT during hospitalization. The incidence of RRT was significantly higher in patients with known CKD (46.4%) compared to those without (28%, p = 0.045). Diabetic nephropathy was the most common etiology and strongly associated with RRT initiation (p = 0.037). Patients requiring RRT presented with markedly elevated serum creatinine, blood urea nitrogen (BUN), phosphorus, LDH, and proteinuria, along with lower hemoglobin, calcium, and platelet levels. Significant correlations were observed between age and both BUN (p = 0.017) and D-dimer (p = 0.007) levels, indicating worse renal and coagulative profiles in older patients. Overall, in-hospital mortality was 10.4%, and although higher among RRT patients, it did not reach statistical significance (p = 0.055).
Pre-existing CKD and diabetic nephropathy were major determinants of RRT initiation among COVID-19 survivors with renal dysfunction. Elevated BUN, creatinine, D-dimer emerged as interrelated markers of renal injury and systemic inflammation. These findings underscore the need for close renal and inflammatory monitoring to prevent irreversible kidney damage in this high-risk population.
