EFFECT OF THE SEMAGLUTIDE IN ISCHEMIC ACUTE KIDNEY INJURY MODEL

https://storage.unitedwebnetwork.com/files/1099/fab7777d96a7dbd22fa546bafd57de48.pdf
EFFECT OF THE SEMAGLUTIDE IN ISCHEMIC ACUTE KIDNEY INJURY MODEL
Guilherme
Henrique Ferreira Vieira
Eloiza de Oliveira Silva eloizaosilva@usp.br University of Sao Paulo School of Nursing Sao Paulo
Carla Djamila de Pina Victoria carlavictoria2001@usp.br University of Sao Paulo School School of Nursing Sao Paulo
Juliana Veloso Gusmão juliana.veloso.silva@usp.br University of Sao Paulo School of Nursing Sao Paulo
Alessandra Oliveira Maia alessandra.omaia@usp.br University of Sao Paulo School of Nursing Sao Paulo
Jessica Paola Garcia Villalba jessica.garcia08@usp.br University of Sao Paulo School School of Nursing Sao Paulo
Maikol Lucas de Camargo Gonçalves maikollcg@usp.br University of Sao Paulo School of Nursing Sao Paulo
Camila Lima camilalima@usp.br University of Sao Paulo School of Nursing Sao Paulo
Bryan Andres Gonçalvez Castaño bryanandres2345@usp.br University of Sao Paulo School of Nursing Sao Paulo
Maria de Fatima Fernandes Vattimo nephron@usp.br University of Sao Paulo School of Nursing Sao Paulo
 
 
 
 
 
 

Semaglutide is an antidiabetic drug analogous to human GLP-1 (glucagon-like peptide), a class of drugs that has recently stood out thanks to its good results in clinical trials, however, the scarcity of data of its effects on chronic and acute cardiovascular and kidney diseases limits its use for the general public. Acute kidney injury (AKI) affects about 49% of patients undergoing major cardiovascular surgeries, in addition to a mortality rate higher than 50%, which justifies the search of new possibilities for the prevention of AKI. ObjectivesTo evaluate the effect of oral semaglutide on renal function in rats submitted to renal ischemia/ reperfusion animal model.

This is a descriptive, prospective, quantitative and experimental study with adult male Wistar rats, weighing 250-300g, randomized into the following groups: SHAM (control, with simulation of renal pedicle clamping); GLP-1 (semaglutide, orally-vo; 3mg/day; 5 days); Ischemia and Reperfusion (IR) (clamping of bilateral renal pedicles for 30 minutes, followed by reperfusion) and GLP-1+IR (semaglutide followed by IR). Renal function was evaluated by inulin clearance (Clin) , serum creatinine (Scr) and urinary flow rate (FU). The oxidative profile was evaluated by urinary peroxides (FOX), lipid peroxides (TBARS) and nitric oxide (NO)

The I/R group showed elevated serum creatinine,  decreased urine flow rate, reduced Clin while the GLP-1+IR group showed increased Clin compared to the I/R group. In addition, the GLP-1+IR group showed a decrease in oxidative metabolites (FOX, TBARS and NO) when compared to IR. 

Table 1; Renal Function

 

Groups

n

Urinary Flow (ml/min)

 

Serum creatinine (mg/dl)

Inulin Clearance (ml/min/100mg)

 

 

 

 

 

 

SHAM

5

    0,013±0,002

 

0,31±0,06

0,81±0,06

GLP-1

5

      0,030±0,013 a

 

0,51±0,1

0,75±0,13

IR

5

      0,011±0,005 b

 

2,27±0,6 ab

0,24±0,03 ab

GLP-1+IR

5

      0,015±0,060 b

 

0,95±0,31 ac

0,51±0,08 ab

 

 

 

 

 

 

 

 

 

 

 

 

Results expressed as mean±standard deviation. SHAM: Control group; GLP-1: Glucagon Like Peptide; I/R: Ischemia and Reperfusion;

a p<0,0001 vs SHAM

b p<0,0001 vs GLP-1

c p<0,0001 vs IR

d p<0,0001 vs GLP-1+IR

 

Table 2. Oxidative and antioxidant profile.

 

Grupos

n

Urinary peroxides (nmol/g de urinary creatinine)

Lipid peroxidation  (mnol/g de urinary creatinine)

Urinary nitrate (nmol/g de urinare creatinine)

 

SHAM

5

3,3±0,6

0,18±0,01

16,0±5,2

 

GLP-1

5

3,2±1,1

0,14±0,04

61,1±25,8a

 

IR

5

15,9±2,8 ab

1,78±0,41 ab

174,5±13,0 ab

 

GLP-1 + IR

5

7,7±2,6 abc

0,20±0,05 c

68,0±20,1 ac

 

 

 

 

 

 

 

 

 

 

 

 

 


Results expressed as mean±standard deviation. SHAM: Control group; GLP-1: Glucagon Like Peptide; IR: Ischemia and Reperfusion;

Semaglutide presented a renoprotective effect associated with its antioxidant role that prevented the reduction in renal function caused by the ischemic AKI.

E-Poster Format Requirements
  • PDF file
  • Layout: Portrait (vertical orientation)
  • One page only (Dim A4: 210 x 297mm or PPT)
  • E-Poster can be prepared in PowerPoint (one (1) PowerPoint slide) but must be saved and submitted as PDF file.
  • File Size: Maximum file size is 2 Megabytes (2 MB)
  • No hyperlinks, animated images, animations, and slide transitions
  • Language: English
  • Include your abstract number
  • E-posters can include QR codes, tables and photos